AIDS: A U.S.- Made Monster? Essay, Research Paper

AIDS: A U.S.- Made Monster?

PREFACE

In an extensive article in the Summer-Autumn 1990 issue of “Top Secret”, Prof J.

Segal and Dr. L. Segal outline their theory that AIDS is a man-made disease,

originating at Pentagon bacteriological warfare labs at Fort Detrick, Maryland.

“Top Secret” is the international edition of the German magazine Geheim and is

considered by many to be a sister publication to the American Covert Action

Information Bulletin (CAIB). In fact, Top Secret carries the Naming Names column,

which CAIB is prevented from doing by the American government, and which names

CIA agents in different locations in the world. The article, named “AIDS: US-

Made Monster” and subtitled “AIDS – its Nature and its Origins,” is lengthy, has

a lot of professional terminology and is dotted with footnotes.

AIDS FACTS

“The fatal weakening of the immune system which has given AIDS its name

(Acquired Immuno-Deficiency Syndrome),” write the Segals, “has been traced back

to a destruction or a functional failure of the T4-lymphocytes, also called

‘helper cells`, which play a regulatory role in the production of antibodies in

the immune system.” In the course of the illness, the number of functional T4-

cells is reduced greatly so that new anti-bodies cannot be produced and the

defenceless patient remains exposed to a range of infections that under other

circumstances would have been harmless. Most AIDS patients die from

opportunistic infections rather than from the AIDS virus itself. The initial

infection is characterized by diarrhea, erysipelas and intermittent fever. An

apparent recovery follows after 2-3 weeks, and in many cases the patient remains

without symptoms and functions normally for years. Occasionally a swelling of

the lymph glands, which does not affect the patient’s well-being, can be

observed.

After several years, the pre-AIDS stage, known as ARC (Aids- Related Complex)

sets in. This stage includes disorders in the digestive tract, kidneys and lungs.

In most cases it develops into full-blown AIDS in about a year, at which point

opportunistic illnesses occur. Parallel to this syndrome, disorders in various

organ systems occur, the most severe in the brain, the symptoms of which range

from motoric disorders to severe dementia and death. This set of symptoms, say

the Segals, is identical in every detail with the Visna sickness which occurs in

sheep, mainly in Iceland. (Visna means tiredness in Icelandic). However, the

visna virus is not pathogenic for human beings. The Segals note that despite the

fact that AIDS is transmitted only through sexual intercourse, blood

transfusions and non- sterile hypodermic needles, the infection has spread

dramatically. During the first few years after its discovery, the number of AIDS

patients doubled every six months, and is still doubling every 12 months now

though numerous measures have been taken against it. Based on these figures, it

is estimated that in the US, which had 120,000 cases of AIDS at the end of 1988,

900,000 people will have AIDS or will have died of it by the end of 1991. It is

also estimated that the number of people infected is at least ten times the

number of those suffering from an acute case of AIDS. That in the year 1995

there will be between 10-14 million cases of AIDS and an additional 100 million

people infected, 80 percent of them in the US, while a possible vaccination will

not be available before 1995 by the most optimistic estimates. Even when such

vaccination becomes available, it will not help those already infected. These

and following figures have been reached at by several different mainstream

sources, such as the US Surgeon General and the Chief of the medical services of

the US Army.

“AIDS does not merely bring certain dangers with it; it is clearly a programmed

catastrophe for the human race, whose magnitude is comparable only with that of

a nuclear war”, say the Segals. ” They later explain what they mean by

“programmed,” showing that the virus was produced by humans, namely Dr. Robert

Gallo of the Bethesda Cancer Research Center in Maryland. When proceeding to

prove their claims, the Segals are careful to note that: “We have given

preference to the investigative results of highly renowned laboratories, whose

objective contents cannot be doubted. We must emphasize, in this connection,

that we do not know of any findings that have been published in professional

journals that contradict our hypotheses.”

DISCOVERING AIDS

The first KNOWN cases of AIDS occurred in New York in 1979. The first DESCRIBED

cases were in California in 1979. The virus was isolated in Paris in May 1983,

taken from a French homosexual who had returned home ill from a trip to the East

Coast of the US. One year later, Robert Gallo and his co-workers at the Bethesda

Cancer Research Center published their discovery of the same virus, which is

cytotoxic. ( i.e poisonous to cells ) Shortly after publishing his discovery,

Gallo stated to newspapers that the virus had developed by a natural process

from the Human Adult Leukemia virus, HTLV-1, which he had previously discovered.

However, this claim was not published in professional publications, and soon

after, Alizon and Montagnier, two researchers of the Pasteur Institute in Paris

published charts of HTLV-1 and HIV, showing that the viruses had basically

different structures. They also declared categorically that they knew of no

natural process by which one of these two forms could have evolved into the

other. According to the professional “science” magazine, the fall 1984 annual

meeting of the American Association for the Advancement of Science (AAAS), was

almost entirely devoted to the question of: to what extent new pathogenic agents

could be produced via human manipulation of genes. According to the Segals, AIDS

was practically the sole topic of discussion.

THE AIDS VIRUS

The Segals discuss the findings of Gonda et al, who compared the HIV, visna and

other closely-related viruses and found that the visna virus is the most similar

to HIV. The two were, in fact, 60% identical in 1986. According to findings of

the Hahn group, the mutation rate of the HIV virus was about a million times

higher than that of similar viruses, and that on the average a 10% alteration

took place every two years. That would mean that in 1984, the difference between

HIV and visna would have been only 30%, in 1982- 20%, 10% in 1980 and zero in

1978. “This means,” say the Segals, “that at this time visna viruses changed

into HIV, receiving at the same time the ability to become parasites in human

T4-cells and the high genetic instability that is not known in other

retroviruses. This is also consistent with the fact that the first cases of AIDS

appeared about one year later, in the spring of 1979.” “In his comparison of the

genomes of visna and HIV,” add the Segals, “Coffin hit upon a remarkable feature.

The env (envelope) area of the HIV genome, which encodes the envelope proteins

which help the virus to attach itself to the host cell, is about 300 nucleotides

longer than the same area in visna. This behaviour suggests that an additional

piece has been inserted into the genomes of the visna virus, a piece that alters

the envelope proteins and enables them to bind themselves to the T4-receptors.

BUT THIS SECTION BEHAVES LIKE A BIOLOGICALLY ALIEN BODY, which does not match

the rest of the system biochemically.

The above mentioned work by Gonda et al shows that the HIV virus has a section

of about 300 nucleotides, which does not exist in the visna virus. That length

corresponds with what Coffin described. That section is particularly unstable,

which indicates that it is an alien object. According to the Segals, it

“originates in an HTLV-1 genome, (discovered by Gallo-ED) for the likelihood of

an accidental occurrence in HIV of a genome sequence 60% identical with a

section of the HTLV-1 that is 300 nucleotides in length is zero.” Since the

visna virus is incapable of attaching itself to human T4 receptors, it must have

been the transfer of the HTLV-1 genome section which gave visna the capability

to do so. In other words, the addition of HTLV-1 to visna made the HIV virus. In

addition, the high mutation rate of the HIV genome has been explained by another

scientific team, Chandra et al, by the fact that it is “a combination of two

genome parts which are alien to each other BY ARTIFICIAL MEANS rather than by a

natural process of evolution, because this process would have immediately

eliminated, through natural selection, systems that are